Research Assistant Professor
Microbiology and Immunology
Microbiology & Immunology
1150 West Medical Center Drive
Ann Arbor, Michigan 48109
[email protected]
1150 West Medical Center Drive
Ann Arbor, Michigan 48109
Available to mentor
Tomoyuki Murakami, PhD
Research Assistant Professor
I study roles of stromal cells in HIV-1 transmission and latency establishment.
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Research InvestigatorUniversity of Michigan Medical School, Microbiology & Immunology, 2023
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Postdoctoral FellowUniversity of Michigan Medical School, Microbiology & Immunology, 2019
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Postdoctoral FellowRIKEN, Wako, 2014
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PhDUniversity of Tokyo, Kashiwa, 2014
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MSUniversity of Tokyo, Kashiwa, 2011
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BSSaitama University, Saitama, 2009
HIV transmission, HIV latency, CD4+ T cells, Stromal cells, Secondary lymphoid organs
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2022 Nov 19;PresentationMacropinocytosis promotes HIV-1 entry into primary CD4+ T cells
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2022 Nov 17;PresentationMechanism of HIV-1 entry and inhibition of HIV-1 entry by host transmembrane proteins
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Sato H, Murakami T, Matsuura R, Abe M, Matsuoka S, Yashiroda Y, Yoshida M, Akari H, Nagasawa Y, Takei M, Aida Y. Viruses, 2022 Jun 16; 14 (6):Journal ArticleA Novel Class of HIV-1 Inhibitors Targeting the Vpr-Induced G2-Arrest in Macrophages by New Yeast- and Cell-Based High-Throughput Screening.
DOI:10.3390/v14061321 PMID: 35746791 -
2022 May 23;PresentationMacropinocytosis promotes HIV-1 entry into primary CD4+ T cells
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Murakami T, Matsuura R, Chutiwitoonchai N, Takei M, Aida Y. Viruses, 2021 Nov 19; 13 (11):Journal ArticleHuntingtin-Interacting Protein 1 Promotes Vpr-Induced G2 Arrest and HIV-1 Infection in Macrophages.
DOI:10.3390/v13112308 PMID: 34835114 -
Murakami T, Ono A. Curr Opin Virol, 2021 Oct; 50: 59 - 68.Journal ArticleHIV-1 entry: Duels between Env and host antiviral transmembrane proteins on the surface of virus particles.
DOI:10.1016/j.coviro.2021.07.005 PMID: 34390925 -
2020 Nov 11;PresentationPSGL-1 and CD43-mediated inhibition of HIV-1 infection and counteraction by HIV-1
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2020 May 21;PresentationPSGL-1 and CD43-mediated inhibition of HIV-1 infection and counteraction by HIV-1